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Health

List Diseases – A

A B C D E F G H I J K L M N O P Q R S T U V W X Y Z
Actinomycosis of the pharynx is a chronic infectious disease of man and animals, caused by the introduction into the pharynx of actinomycetes (radiant fungal parasites).
In classical works, the fungus Actynomyces bovis was used as the pathogen, but from the latest works of the Romanian otorhinolaryngologist V.Racovenu (1964) it follows that Actinomyces Israeli is the true causative agent of actinomycosis.
Actinomycosis of the esophagus is a rare disease, the occurrence of which is possible only if the mucosa of the esophagus is damaged and the infected agents stay in it.
Actinomycosis is a chronic infectious disease of humans and animals caused by anaerobic radiant fungi.
Actinitic keratosis (syn: senile keratosis, solar keratosis) develops as a result of prolonged exposure to ultraviolet rays on exposed skin areas, usually in persons over 50 years old.
The actinic reticuloid was first described and isolated in a separate nosological unit in 1969. FA Ive et al. This disease in the literature is described under the name of chronic actinic dermatitis.
Actinic elastosis (elastoidosis) occurs with prolonged exposure to ultraviolet rays, usually observed in senile age (senile elastosis).
Actinic cheilitis is a disease caused by the increased sensitivity of the red border to ultraviolet radiation (allergic reaction of delayed type), one of the symptoms of photodermatosis.
Symptoms of acromegaly usually appear after 20 years, develop gradually. Early signs are swelling and hypertrophy of the soft tissues of the face and limbs. The skin thickens, the severity of the skin folds increases. Increasing the volume of soft tissues makes it necessary to constantly increase the size of shoes, gloves, rings.
Acromegaly and gigantism are related to neuroendocrine diseases, which are based on a pathological increase in growth activity.
Acrokeratosis vertexiform Gopfa - genodermatosis with autosomal dominant type of inheritance. Sometimes occurs in combination with Daria's disease, which, according to some authors, is an expression of a birth defect in keratinization.
Family acroheria (Gottron's syndrome) is a rare disease described in 1941 by N. Gottron. The causes and pathogenesis of the acroheria of the family (Gottron's syndrome) are not fully understood. In the development of the disease, an important role is played by the disruption of the structure and function of fibroblasts and collagen synthesis, hypofunction of the pituitary gland. There are reports of family cases of the disease.
Acrodermatitis resistant pustular Allopo (synonyms: acropustulosis, Crocker's persistent dermatitis) is a chronic relapsing disease characterized by the defeat of the coccyxes of the fingers and toes, on which there are pustular eruptions prone to spread.
Acrodermatitis of papular children (synovial syndrome of Gianotti-Crosti) is an acute disease, the development of which is associated with the hepatitis B virus, less often with other viral infections.
Acrocyanosis is a permanent, painless, symmetrical cyanosis of the hands, feet, or face caused by the vascular spasm of small skin vessels in response to cooling.
Acquired primary hypogonadism can have a different genesis. It can be the outcome of infectious-inflammatory lesions of testicles and / or their appendages.
Acquired platelet dysfunction may be the result of taking aspirin, other non-steroidal anti-inflammatory drugs, or systemic diseases.
Primary acquired hypothyroidism develops as a result of endemic iodine deficiency, autoimmune thyroiditis, thyroid gland surgery, inflammatory and tumor thyroid diseases, uncontrolled therapy with thyreostatic drugs in thyrotoxicosis.
Acute acoustic trauma occurs as a result of the impact on the hearing organ of a powerful impulse noise of more than 160 dB, often in combination with a sharp increase in the barometric pressure in the explosion.
Acoustic trauma occurs as a result of prolonged or impulsive noise on the organ of hearing or vibration, exceeding the permissible norms or tolerance of the receptor structures of the inner ear to these stimuli.

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