List Diseases – M
Munchhausen's syndrome - a severe and chronic form of imitation of the disease - consists in the repeated production of false physical symptoms in the absence of external benefit; the motivation for this behavior is to assume the role of the patient
To date, the embryogenesis of sebocystomatosis has not been fully understood and remains a matter of controversy. A hundred years ago, many dermatologists regarded lesions as fatty or retention cysts. Some authors believe that cysts are formed as a result of excessive keratinization leading to a delay in the secretion of sebaceous glands.
Multiple sclerosis is the most common cause of the acquired demyelination of the central nervous system, which, in fact, is an inflammatory process directed against the myelin of the brain and spinal cord.
Multiple pregnancy is a pregnancy in which two or more fetuses develop in a woman's body. Genera of two or more fruits are called pluriparous.
Multiple organ failure is a pathological condition characterized by a disruption in the adaptation reaction of the organism. This condition is accompanied by serious metabolic disorders
Multiple myeloma (myelomatosis; plasma cell myeloma) is a plasma cell tumor that produces a monoclonal immunoglobulin that implants and destroys nearby bones.
In clinical practice, the greatest difficulties for diagnosis are endocrine diseases with symptoms of impaired functions of several endocrine glands. In most cases, clinical features of this kind are manifested in hypothalamic-pituitary disorders.
Multiple endocrine neoplastic syndrome, or Type I MEN (multiple type I endocrine adenomatosis, Vermeer's syndrome), is a hereditary disease characterized by tumors in the parathyroid glands, in the pancreas and in the pituitary gland. Clinical manifestations are expressed by hyperparasitism and asymptomatic hypercalcemia.
Multiple endocrine neoplastic syndrome IIA type (MEN type IIA syndrome, multiple endocrine adenomatosis, type IIA syndrome, Sipple syndrome) is a hereditary syndrome and is characterized by medullary thyroid cancer, pheochromocytoma and hyperparathyroidism. The clinical picture depends on the affected glandular elements.
The hormone-active pancreatic tumor can be one of the manifestations of multiple endocrine adenomatosis (MEA) or multiple endocrine neoplasia (MEN).
Multiple endocrine neoplastic syndrome, IIB type (MEN IIB, MEN type II syndrome, neuroma mucosa syndrome, multiple endocrine adenomatosis) is characterized by multiple mucous neuromas, medullary thyroid carcinomas, pheochromocytomas and often Marfan syndrome.
Multiple chemical sensitivity syndrome (idiopathic environmental intolerance) is characterized by current, uncertain signs attributed to exposure to chemically unbound substances of low level, commonly found in the environment.
The causative agent of the disease is Mallasseria furfur. Multicolored lichen occurs as a result of transformation of the saprophilic form into a pathogenic form or infection from the outside. The development of multi-colored hair loss is facilitated by the weakening of the immune system, increased sweating, endocrine disorders.
Mukocele of paranasal sinuses is a peculiar retentional saccular cyst of any one paranasal sinus, formed as a result of obliteration of the nasal excretory duct and accumulation of mucous and hyaline secretions inside the sinus, as well as elements of desquamation of the epithelium
Mukocele develops when the drainage of the normal sinus secretion is disturbed due to infection, allergies, trauma or congenital narrowness of the drainage pathways.
Mucous-cutaneous lymphonodular syndrome (acute children's feverish cutaneous mucocutaneous glandular syndrome, Kawasaki's disease, Kawasaki syndrome) is an acute systemic disease characterized by a morphologically predominant lesion of the middle and small arteries with the development of destructive-proliferative vasculitis identical to nodular polyarteritis, and clinically - fever, changes in mucous membranes, skin, lymph nodes, possible damage to coronary and other visceral arteries.
Mucopolysaccharidosis VII is an autosomal recessive progressive disease resulting from a decrease in the activity of lysosomal beta-D-glucuronidase, which is involved in the metabolism of dermatan sulfate, heparan sulfate, and chondroitin sulfate.
Mucopolysaccharidosis, type VI is an autosomal recessive disease characterized by severe or mild clinical symptoms; is similar to the Hurler syndrome, but differs from it with a reserved intellect.
Mucopolysaccharidosis, type IX is an extremely rare form of mucopolysaccharidosis. To date, there is a clinical description of one patient, a girl of 14 years.
Mucopolysaccharidosis IV is an autosomal recessive progressive genetically heterogeneous disease that occurs as a result of mutations in genes encoding galactose-6-sulphatase (N-acetylgalactosamine-6-sulfatase) that participates in the metabolism of keratan sulfate and chondroitin sulfate or in the beta-galactosidase gene (this form is an allelic variant of Gml-gangliosidosis) leading to the manifestation of mucopolysaccharidosis IVA and mucopolysaccharidosis IVB, respectively.