Diseases of children (pediatrics)

What triggers nodular polyarteritis?

Nodular polyarteritis is referred to polyethological diseases. Possible causes of its occurrence may be infectious factors, medications, vaccination. In classic nodular polyarteritis, most patients are diagnosed with hepatitis B virus infection. In juvenile polyarteritis, the onset of the disease and its aggravation coincide with a respiratory viral infection, tonsillitis or otitis, less often with a drug or vaccination provocation.

Nodular polyarteritis

Nodular polyarteritis (Kussmaul-Meyer's disease, classic nodular polyarteritis, nodular polyarteritis with predominant lesion of internal organs, nodular polyarteritis with predominant involvement of peripheral vessels, nodular polyarteritis with leading thrombangiotic syndrome) - acute, subacute or chronic disease, which is based on damage to peripheral and visceral arteries.

Hemorrhagic vasculitis in children

Shenlaine-Henoch disease (hemorrhagic vasculitis, anaphylactoid purpura, hemorrhagic vasculitis, allergic purpura, haemorrhagic purpura Genocha, capillarotoxicosis) is a widespread systemic disease with a predominant lesion of the microcirculatory bed of the skin, joints, gastrointestinal tract, kidneys.

How are systemic vasculitis treated?

Treatment of an active (acute) period of systemic vasculitis should be performed in a specialized (rheumatological) hospital, when remission is reached, the patient should continue treatment on an outpatient basis, under the supervision of a pediatrician, a rheumatologist and, if necessary, narrow specialists.

What provokes systemic vasculitis?

Systemic vasculitis develops in children with altered reactivity. Among the factors contributing to their occurrence are most significant: frequent acute infectious diseases, foci of chronic infection, drug allergy, hereditary predisposition to vascular or rheumatic diseases.

Systemic vasculitis

Systemic vasculitis is a heterogeneous group of diseases, the main morphological feature of which is inflammation of the vascular wall, and the spectrum of their clinical manifestations depends on the type, size, localization of the affected vessels and the severity of the associated inflammatory changes.

Amyloidosis

Amyloidosis is a violation of protein metabolism, accompanied by the formation in the tissues of a specific protein-polysaccharide complex (amyloid) and the defeat of many organs and systems.

Juvenile ankylosing spondylitis

Juvenile spondyloarthritis is a group of clinically and pathogenetically similar rheumatic diseases of childhood, including juvenile ankylosing spondylitis, juvenile psoriatic arthritis, reactive (post-enterocolitis and urogenital) arthritis associated with HLA-B27 antigen, Reiter's syndrome, enteropathic arthritis in inflammatory bowel diseases (regional enteritis , ulcerative colitis).

Reactive arthritis in children

Reactive arthritis is an aseptic inflammatory joint disease that develops in response to extra-articular infection; the putative primary agent can not be isolated from the joints using conventional artificial nutrient media.

Treatment of juvenile chronic arthritis

During periods of exacerbation of juvenile rheumatoid arthritis, the infant's movement should be limited. Complete immobilization of joints with overlapping of langets is contraindicated, it contributes to the development of contractures, muscle tissue atrophy, aggravation of osteoporosis, rapid development of ankylosis. Physical exercises contribute to the preservation of the functional activity of the joints. Useful cycling, swimming, walking. Running, jumping, active games are undesirable.

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