This form of the disease of the ear maze was first described by P. Menier in 1848 by a young woman who, traveling in a stagecoach in winter, suddenly became deaf in both ears, she also had dizziness and vomiting.
Timpanosclerosis is characterized by cicatrical and degenerative manifestations in the middle ear, caused by the previous inflammatory-destructive process, culminating in scar tissue formation.
According to VT Palchun et al. (1977), sigmoid and transverse sinuses are most commonly affected (79%), then the jugular vein bulb (12.5%), the remaining cases occur in cavernous and stony sinuses.
Syphilitic lesions of the ear maze are characterized by a complex pathogenesis, some aspects of which remain unexplored to the present day. Many authors treat these lesions as one of the manifestations of neurosyphilis (neirolabirintit), caused by syphilitic changes in the liquid media of the inner ear (similar to changes in syphilis in the cerebrospinal fluid).
Vestibular neuronitis - an acute (viral) lesion of the vestibular ganglion, vestibular nuclei and other retrolabirint structures, isolated into an independent nosological form in 1949 by the American otolaryngologist by C.Hallpike.
Acute infectious diseases, especially in children, are often the cause of severe damage to the inner ear, leading to partial or complete deafness, imperfection of the function of the vestibular apparatus.
According to summary statistics of the second half of the 20th century, 98% of purulent diseases of the cerebellum occur in the otogenous abscess of the cerebellum.
Primary syphilis manifested by chancre is very rare and occurs as a result of accidental infection of the auricle or external auditory canal with skin damage or with a kiss.
Primarily occurs exceptionally rarely. As a rule, tuberculous otitis media develops against the background of pulmonary tuberculosis or bones. Patients begin to notice a one- or two-sided hearing loss, accompanied by ear noise.
According to the materials of the Romanian author I.Tesu (1964), most often otomastoiditis in infants occurs at the age of up to 6 months after birth, after which it exponentially decreases to the frequency of occurrence in adults.
