Recommendations for monitoring the health of children with achondroplasia have been set out by the Genetics Committee of the American Academy of Pediatrics. These recommendations serve as a guide and do not replace individual decision making. A recent review [Pauli & Botto 2020] also includes guidance from a guide. There are specialized clinics for the treatment of skeletal dysplasia; their recommendations may differ slightly from these general recommendations.
Recommendations include (but are not limited to) the following.
Hydrocephalus. If signs or symptoms of increased intracranial pressure appear (for example, accelerated head growth, a constantly bulging fontanelle, a noticeable increase in the protrusion of superficial veins on the face, irritability, vomiting, changes in vision, headache), a referral to a neurosurgeon is necessary.
The assumed etiology of hydrocephalus in achondroplasia is increased intracranial venous pressure due to stenosis of the jugular foramen. Thus, ventriculoperitoneal shunting was the standard treatment. However, endoscopic third ventriculostomy may be beneficial in some people, implying that other mechanisms, such as blockage of the ventricular outlets due to craniocervical stenosis, may be of value. 
Narrowing of the craniocervical junction. The best predictors of the need for suboccipital decompression are:
- Hyperreflexia or clonus of the lower extremities
- Central hypopnea on polysomnography
- Reduction in the size of the foramen magnum, determined by computed tomography of the craniocervical junction and compared with the norms for children with achondroplasia. 
- Signs of spinal cord compression and / or T 2 weighted signal abnormalities; has recently been suggested as another factor that should be considered when deciding to work.
If there are clear signs of symptomatic compression, you should urgently contact a pediatric neurosurgeon for decompression surgery. 
Obstructive sleep apnea. Treatment may include the following:
- Positive airway pressure
- Tracheostomy in extreme cases
- Reducing weight
These interventions can lead to improved sleep disturbances and some improvement in neurological function. 
In the rare cases where the obstruction is severe enough to warrant a tracheostomy, surgery to advance the midface has been used to relieve upper airway obstruction. 
Dysfunction of the middle ear. If necessary, aggressive treatment of frequent middle ear infections, persistent fluid in the middle ear, and subsequent hearing loss should be undertaken. Long-acting tubes are recommended because they are often needed up to seven or eight years of age. 
If problems arise at any age, it is recommended to apply appropriate treatment methods.
Low stature. A number of studies have evaluated growth hormone (GH) therapy as a possible treatment for short achondroplasia. 
In general, these and other series show an initial acceleration of growth, but its effect decreases over time.
On average, you can expect an increase in the height of an adult by only about 3 cm.
Extended limb lengthening using various techniques remains an option for some. You can increase your height up to 30-35 cm.  Complications are frequent and can be serious.
While some advocate performing these procedures as early as six to eight years of age, many pediatricians, clinical geneticists, and ethicists advocate postponing such surgery until the young person can participate in an informed decision.
In North America at least, only a small proportion of affected individuals choose to undergo extended limb lengthening. The Little People of America Medical Advisory Board has released a statement regarding the use of extended limb lengthening.
Obesity. Measures to prevent obesity should start early in childhood. Standard treatments for obesity should be effective in people with achondroplasia, although the calorie requirements are lower. 
Standard weight and weight-for-height grids specific for achondroplasia should be used to track progress. It is important to note that these curves are not ideal weight-for-height curves; they were obtained from thousands of data points from people with achondroplasia.
Body mass index (BMI) standards have been developed for children aged 16 and under. BMI is not standardized for adults with achondroplasia; comparison with BMI curves for average height will give incorrect results. 
Deformation of the varus. Annual orthopedic follow-up is recommended by either a provider familiar with achondroplasia or an orthopedic surgeon. Surgical criteria published. 
The presence of progressive symptomatic bending requires referral to a podiatrist. The deformity of the varus itself without symptoms usually does not require surgical correction. Various interventions can be selected (eg, guided growth using eight plates, valgus osteotomy and derotation osteotomy). There are no controlled studies comparing the outcome of treatment options.
Kyphosis. Infants with achondroplasia often develop flexible kyphosis. A protocol is available to help prevent the development of fixed angular kyphosis, which includes avoiding flex-back strollers, swings, and carrying. Council against sitting without support; always apply back pressure when holding your baby.
- Kyphosis improves or goes away in most children after orthograde and walking. 
- In children who do not have spontaneous remission after increasing torso strength and starting to walk, fixation is usually sufficient to prevent thoracolumbar kyphosis from persisting. 
- If severe kyphosis persists, spinal surgery may be required to prevent neurological complications. 
Spinal stenosis. If serious signs and / or symptoms of spinal stenosis develop, an urgent referral to a surgical specialist is needed.
Wide and wide laminectomy is usually recommended. The relevance of the procedure depends on the level (eg, thoracic or lumbar) and the degree of stenosis. Patients had better outcomes and improved function than they had previously undergone surgery after onset of symptoms 
Immunization. Nothing about achondroplasia rules out all routine vaccinations. Given the increased respiratory risk, DTaP, pneumococcal and influenza vaccines are especially important.
Adaptive needs. Due to the small growth, environmental changes are necessary. In school, this may include stools, lowered light switches, toilets of appropriate heights or other means of access, lower desks, and leg rests in front of chairs. All children should be able to leave the building on their own in the event of an emergency. Small hands and weak ligaments can make fine motor skills difficult. Appropriate adaptations include the use of smaller keyboards, heavier nibs, and smoother writing surfaces. Most children must have an IEP or 504.
Pedal extensions are almost always needed for riding. Workspace modifications such as lower desks, smaller keyboards, steps and toilet access may also be required.
Socialization. Due to the very noticeable short stature associated with achondroplasia, sick people and their families may find it difficult to socialize and adapt to school.
Support groups such as Little People of America, Inc. (LPA) can help families address these issues through peer support, example, and social awareness programs.
Information on employment, education, the rights of persons with disabilities, adoption of short children, medical problems, appropriate clothing, adaptive accommodations and parenting is available through the national newsletter, seminars and workshops.
There are no medications, as well as non-medicinal agents, that can cure this congenital malformation.
The most commonly used physiotherapy treatment; may also require treatment for hydrocephalus (bypass grafting or endoscopic ventriculostomy), obesity, sleep apnea, middle ear infection, or spinal stenosis.
In some clinics, after the child is five to seven years old, they undertake surgical treatment: lengthening the bones of the lower leg, thighs and even the humerus or correcting deformities - using operations and special orthopedic devices - in three to four stages, with a duration of each up to 6-12 months...
Research phase therapy
The introduction of a C-type natriuretic peptide analogue is undergoing clinical trials. Early results showed that it was well tolerated and resulted in an increase in growth rate from baseline in children with achondroplasia ( trial site ). The conjugated C-type natriuretic peptide is also currently in clinical trials (trial site ) Other considerations include tyrosine kinase inhibition , meclizine and soluble recombinant human FGFR3 decoy. 
Search clinicaltrials.gov in the US and EU Clinical Trials Registry in Europe for information on clinical trials for a wide range of diseases and conditions.