Purulent complications of angina
Last reviewed: 23.04.2024
All iLive content is medically reviewed or fact checked to ensure as much factual accuracy as possible.
We have strict sourcing guidelines and only link to reputable media sites, academic research institutions and, whenever possible, medically peer reviewed studies. Note that the numbers in parentheses ([1], [2], etc.) are clickable links to these studies.
If you feel that any of our content is inaccurate, out-of-date, or otherwise questionable, please select it and press Ctrl + Enter.
Local purulent complications of angina and in the neighborhood - acute otitis media, acute laryngitis, laryngeal edema, phlegmon of the neck, parapharyngeal abscess, acute cervical lymphadenitis, lesions of the salivary glands (sialoadenitis). To complications at a distance should include arthritis and arthrosis, orchitis, cholecystitis, meningitis, etc.
To general purulent complications of angina, apparently, should be attributed to general toxic syndrome with a violation of the central nervous system and cardiovascular system, as well as post-genital septicemia. Frequent recurrent tonsillitis promotes sensitization of the body, against which the general resistance of the organism to other infectious diseases decreases.
Postaginous septicemia is a severe purulent complication of angina and is currently extremely rare. Most often, it occurs in people who suffer angina between the ages of 16 and 35 years. In severe cases, this complication can be fatal. Post-anginal septicemia is divided into primary and secondary. As a rule, primary septicemia occurs after the transferred banal angina and relate to monobacillary complications: anaerobes, saprophytes of the oral cavity (B. Funduliformis, gram-negative bacterium very sensitive to penicillin, B. Fragilis, B. Ramosus, etc.). Secondary septicemia occurs as a complication of ulcerative necrotic tonsillitis, for example, with agranulocytosis or leukemia. They proceed much heavier than primary and are related to the polyangiogenic complications of angina. The entrance gates for microorganisms are the internal jugular vein or cavernous sinus, in which the infection penetrates due to a violation of the barrier function of the vascular wall in follicular and lacunar angina or peritonsillar phlegmon. The infected thrombus originating in the vein is the source of septicemia, and in some cases, when purulent emboli are separated, pyemia. In the latter case, metastatic abscesses occur at a distance (in the lungs, joints, liver, etc.).
Latent period of post-genital septicemia from 1 to 15 days. The clinical course is characterized by a sharp rise in body temperature, severe chills, accompanied by a "cold" sweat, a weak cardiovascular activity (frequent threadlike pulse, a sharp decrease in blood pressure, hypoxia) that occurred suddenly either in the recovery period, or some time after it full of health. The face of the patient becomes gray with an icteric shade. With a high body temperature and severe clinical course, the patient periodically falls into an unconscious state, raves. Sopor and coma are harbingers of a lethal outcome, which in the case of superheavy flow can occur within 3 days. In the acute course of postaginous septicemia, death can occur 5-10 days after the onset of the disease. Purulent complications of subacute angina are cured by the appointment of large doses of penicillin.
[