Out of exogenous effects, the causes of development of iridocyclitis can be concussions, burns, injuries, which are often accompanied by the introduction of infection.
The clinical picture of inflammation distinguishes between serous, exudative, fibrinous, purulent and hemorrhagic iridocyclitis, acute and chronic in nature of the flow, focal (granulomatous) and diffuse (non-granulomatous) forms of inflammation in the morphological pattern. Focal pattern of inflammation is typical for hematogenous metastatic infection.
The morphological sub-extract of the main focus of inflammation in granulomatous iridocyclitis is represented by a large number of leukocytes, there are also mononuclear phagocytes, epithelioid, giant cells and necrosis zone. From such a focus can be identified pathogenic flora.
Infectious-allergic and toxic-allergic iridocyclitis occur in the form of diffuse inflammation. In this case, the primary damage to the eye can be outside the vascular tract and located in the retina or optic nerve, from where the process spreads to the anterior section of the vascular tract. In those cases when the toxic-allergic lesion of the vascular tract is primary, it never has the character of a real inflammatory granuloma, but arises suddenly, develops rapidly as a hyperergic inflammation.
The main manifestations are a violation of microcirculation with the formation of fibrinoid swelling of the vascular wall. In the focus of the hyperergic reaction there are edema, fibrinous exudation of the iris and ciliary body, plasma lymphoid or polynucleic infiltration.
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