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Acute simple adenoiditis
Last reviewed: 23.04.2024
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Acute simple adenoiditis, or retinasal angina, is the inflammation of adenoid vegetation, which occurs most often in early childhood and in the first years of life. Manifestations of this disease in early childhood (up to 1 year of life) and in later ages are different. There are also acute or subacute recurrent and prolonged adenoiditis.
Symptoms of acute simple adenoiditis
Acute simple adenoiditis in children begins suddenly with a rise in body temperature to 40-41 ° C, often accompanied by convulsive syndrome, spasm of the larynx, frequent breathing, tachycardia and arrhythmia. The ill infant refuses to breast because of the impossibility of sucking (lack of nasal breathing), which quickly leads to a decrease in the body weight of the child. When pharyngoscopy is determined by the flow of pus, but the back wall of the pharynx, which the child swallows. Submandibular lymph nodes are enlarged and painful on palpation. In the predominantly one-sided lesion of the nasopharyngeal tonsil, the lymph nodes that are enlarged on one side interfere with the function of the thoracic-clavicular-mastoid muscle, which causes the involuntary position of the head, which is somewhat turned to the sore and lowered. With otoscopy, the tympanic membrane can be defined. The increase in body temperature can last from 3 to 5 days. Complications that can occur with acute simple adenoiditis include acute inflammation of the upper respiratory tract (laryngotracheitis), bronchopneumonia, acute otitis, parapharyngeal abscesses and phlegmon, which makes the forecast very cautious.
Acute simple adenoiditis in childhood also has an acute onset and is often accompanied by streodorous laryngitis, otalgia, meningism, hypoxia. Absent nasal breathing is compensated by breathing through the mouth. There is a closed nasal.
With anterior and posterior rinoscopy, abnormally enlarged, hyperemic or pseudo-adenoidal growths are found, covering the chorines (with posterior rinoscopy) and prolapse in the posterior-superior parts of the nasal cavity (with anterior rhinoscopy). On the back wall of the pharynx, purulent discharge flows, which are also determined in the nasal cavity. Usually there is also an associated palatine amygdalite.
Recurrent adenoiditis in children, which usually occur during the cold season, are very frequent. This form of acute simple adenoiditis, beginning with early childhood, leads to an ever greater relapse of adenoid tissue with each new relapse, which is accompanied by impaired development of the facial skull, bite deformities and other undesirable consequences in the development of the child.
Complications with this form of acute simple adenoiditis are numerous (otitis, sinusitis, adeophlegmons, diseases of the lower respiratory tract, etc.). Such a child sharply lags behind in development from his peers.
Acute prolonged adenoiditis differs from acute simple adenoiditis by longer development and clinical course (several weeks). There is some discrepancy between the increased body temperature and the relatively satisfactory state of the child. Nasal breathing can be satisfactory, breastfeeding the baby does not cause any particular "difficulties. Endoscopic signs of the disease are less pronounced than with acute simple adenoiditis.
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How is acute acute adenoiditis recognized?
The diagnosis of acute simple adenoiditis is established on the basis of the clinical picture and inflammatory changes in the nasopharyngeal tonsil. In all cases, this form of adenoiditis should be differentiated from diphtheria by bacteriological examination of the smear from the nasopharynx and palatine tonsils.
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Treatment of acute simple adenoiditis
Treatment of acute simple adenoiditis in infants should be directed primarily to the restoration of nasal breathing, at least for the period of feeding. In the rest, the treatment is performed as in follicular angina with the appointment of antibiotics and under the supervision of a pediatrician. With prolonged acute adenoiditis, European otorhinolaryngologists produce adenotomy in the "warm" period followed by intensive penicillin therapy. Also recommend removal of adenoids in the event of a toxic syndrome or in the ineffective course of auricular complications. If the child even once suffered an acute adenoiditis, it is advisable to carry out adenotomy, since in the vast majority of cases acute acute adenoiditis inevitably passes into chronic adenoiditis with manifestations of the syndrome of focal infection.
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