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Glaucoma - Top 100

Pigment Dispersion Syndrome (SPD) is a condition in which the pigment is washed out of the pigment epithelium of the back of the iris, its subsidence on various structures of the anterior segment of the eye. Obstruction and subsequent destruction of the trabecular network can result in increased intraocular pressure and the development of secondary open-angle glaucoma.
Exfoliative syndrome is a systemic disease that leads to the development of secondary open-angle glaucoma.
Secondary open-angle glaucoma can develop in almost any mode of administration of glucocorticoid drugs. Elevation of intraocular pressure can be pronounced and prolonged.
Heterochromic iridocyclitis Fuchs is a one-sided chronic inactive non-granulomatous anterior uveitis associated with secondary posterior subcapsular cataract and glaucoma in 13-59% of cases.
Syphilis of the eye can be a congenital or acquired disease, transmitted sexually. In congenital syphilis, as a rule, the anterior segment of the eye is affected with the development of interstitial keratitis and anterior uveitis, and with acquired syphilis, both anterior and posterior uveitis develop.
Juvenile rheumatoid arthritis is a common cause of uveitis in children, which often develop an increase in intraocular pressure and glaucoma.
Upon penetration of the lens proteins through an intact or damaged capsule, a strong intraocular inflammatory reaction is triggered into the anterior chamber of the eye or vitreous cavity, as a result of which an outflow of intraocular fluid may occur with the development of an acute increase in intraocular pressure or glaucoma.
Sarcoidosis is a systemic disease characterized by the formation of noncaseating, granulomatous inflammatory infiltrates in the lungs, skin, liver, spleen, central nervous system and eyes.
Facial glaucoma develops with mature or overripe cataracts. When percolation of soluble lens proteins into the anterior chamber, the trabecular network is blocked, leading to an increase in intraocular pressure.
Glaucoma of lens masses develops when the integrity of the lens capsule is broken and its cortex and proteins are released into the anterior chamber. This situation occurs after extracapsular extraction of cataracts, lens trauma with a capsule rupture and neodymium IAG laser back capsulotomy, in which free lens particles block the trabecular network, disrupting the outflow of watery moisture.

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